Type 1 diabetes (T1D) is an autoimmune condition in which the body's immune system mistakenly attacks and destroys insulin-producing beta cells in the pancreas. This results in an inability to regulate blood sugar levels. Although often diagnosed in children or adolescents, T1D can occur at any age. Insulin therapy and lifestyle management are critical for long-term survival and health.
Although T1D results primarily from genetic and autoimmune processes, certain environmental factors may influence its onset.
Viral infections (e.g., enteroviruses) can trigger autoimmune responses in genetically predisposed individuals.
Family history increases susceptibility, especially with certain HLA gene mutations.
Early exposure to cow's milk protein or gluten during childhood has been suggested as a potential trigger in some cases.
T1D symptoms often develop rapidly and require immediate medical attention.
Excessive fluid consumption due to dehydration.
Caused by high levels of glucose in the blood.
Due to the breakdown of fat and muscle to produce energy.
The result of the body's inability to use glucose as an energy source.
High glucose levels affect the balance of fluids in the eyes.
An accurate and timely diagnosis is essential for managing T1D and preventing complications.
Normal
<60 mg/dL.
Diabetic
>126 mg/dL
Reflects the average blood sugar level over three months.
Normal
< 5.7%
Diabetic
> 6.5%
Identifies immune markers that target beta cells.
Differentiate T1D from T2DM by measuring insulin production.
Insulin therapy is the cornerstone of T1D management, along with lifestyle interventions.
Administered through insulin injections or pumps to maintain blood glucose levels.
Tracks glucose levels in real time for better control
Improves glucose absorption and reduces insulin requirements.
Emphasizes carbohydrate counting and foods with a low glycemic index.